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Cell death pathways in photodynamic therapy of cancer
Pawel Mroz
, Anastasia Yaroslavsky
, Gitika B. Kharkwal
,
Michael R. Hamblin
Massachusetts General Hospital
Harvard University
Boston University College of Engineering
Harvard-MIT Division of Health Sciences and Technology
Research output
:
Contribution to journal
›
Review article
›
peer-review
639
Citations (Scopus)
Overview
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Biochemistry, Genetics and Molecular Biology
Photodynamics
100%
Cell Death
100%
Mitochondrion
40%
Light
20%
Lipid Metabolism
20%
Alpha Oxidation
20%
Lysosome
20%
Calcium Metabolism
20%
Enzyme
20%
Cytokine
20%
Mediator
20%
Autophagy
20%
Cytochrome
20%
Transcription Factors
20%
Endoplasmic Reticulum Stress
20%
Caspase
20%
Bcl-2
20%
Protein Kinases
20%
Acute Stress
20%
Golgi Apparatus
20%
Proteasome
20%
Immunology and Microbiology
Cell Death
100%
Photodynamics
100%
Mitochondrion
40%
Blood Plasma
20%
Light
20%
Mediator
20%
Autophagy
20%
Caspase
20%
Endoplasmic reticulum
20%
Cytokine
20%
Transcription Factors
20%
Lipid Metabolism
20%
Lysosome
20%
Endoplasmic Reticulum Stress
20%
Proteasome
20%
Acute Stress
20%
Calcium Metabolism
20%
Bcl-2
20%
Pharmacology, Toxicology and Pharmaceutical Science
Photodynamic Therapy
100%
Malignant Neoplasm
100%
Photosensitizer
40%
Reactive Oxygen Metabolite
20%
Protein
20%
Cytokine
20%
Neoplasm
20%
Caspase
20%
Protein Kinases
20%
Transcription Factors
20%
Cytochrome C
20%
Proteasome
20%
Keyphrases
Oxidized Proteins
25%
Induced Oxidation
25%
ER Stress Response
25%